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In summary, GSH enables antibiotic inactivation by serving as a substrate for detoxifying enzymes, such as FosA, and by modulating the expression or stability of antibiotic-degrading enzymes, including the downregulation of NDM-1

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That contrast, targeted signaling versus systemic support, is the heart of the NAD+ vs peptides question, and it is why the two are so often better together than in competition