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Tom Kensler and colleagues have provided compelling evidence that Notch1 contributes to the diminished capacity to regenerate liver in Nrf2-null mice based on the fact that the proximal region of the Notch1 promoter contains an ARE sequence (5′-CTGAGCGGGCG-3′) and that Nrf2 −/− MEF cells exposed to Notch1 signaling ligands display reduced expression of Notch1, p21, Hes1, Herp1, and Nrarp compared with wild-type fibroblasts treated in a similar manner [135]

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Oxidative stress is intimately linked to a variety of pathophysiological processes, including neuroinflammation, dysfunction of mitochondria and autophagical disorder

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