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As a result, two things happen with B12 deficiency: Homocysteine levels rise because there is less THF available for the reaction that converts homocysteine to methionine and MMA levels rise because vitamin B12 is required to convert methylmalonyl-CoA to succinyl-CoA

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The suppression of hyperlipid diet-induced ferroptosis of vascular smooth muscle cells protests against atherosclerosis independent of p53/SCL7A11/GPX4 axis